Category: Immune Support

Vitamin E


The Premier Fat-Soluble Antioxidant

Vitamin E is the primary fat-soluble antioxidant vitamin in the body. (Vitamin C is the primary water-soluble antioxidant). Vitamin E plays a major role in  cellular respiration. Deficiencies of Vitamin E are associated with:

  • heart disease
  • cancer
  • strokes
  • arthritis
  • allergies
  • infections
  • inflammation
  • diabetes
  • neurological damage
  • muscle weakness
  • fibrocystic breast disease
  • eczema
  • macular degeneration
  • poor wound healing

Dietary sources of Vitamin E include: wheat germ oil, nuts, whole grains, egg yolk.

NOTE: Doses over 800 IU per day of vitamin E may elevate triglycerides.

Maxi Multi provides 400 IU per day of Vitamin E

Those requiring additional Vitamin E supplementation should consider Tocotrienols

Zinc


The Enzyme Activator

Zinc is a mineral that functions as a co-factor in numerous metabolic processes. In fact, zinc is a co-factor in over 200 enzymes in the body.

Zinc deficiency is associated with:

  • prostate enlargement
  • immune deficiency
  • atherosclerosis
  • malabsorption syndromes
  • slow wound healing
  • loss of taste or smell
  • impaired glucose tolerance
  • skin disorders of every type
  • Rheumatoid Arthritis (RA)
  • inflammatory bowel disease (IBS)
  • abnormal menstruation

The adult daily dose range is from 15-50 mg.

Food sources include wheat germ, wheat bran, pumpkin seed, avocado, sea food.

Caution: Large doses (more than 50mg/day) can cause a copper deficiency & other mineral imbalances. Copper should be supplemented when using zinc in high doses.

Optimal daily amounts of Zinc are easily obtained from Dr. Myatt’s Maxi Multi – a comprehensive multiple vitamin and mineral nutrient formula. Click here for full information.

 

Vitamin C

Master Immune Stimulant and Antioxidant

Vitamin C is a water-soluble vitamin that plays a major role in numerous biological functions including:

  • collagen synthesis (production of tendon, ligament, cartilage and skin)
  • immune function – increases white blood cell activity, interferon production and thymic hormone secretion.
  • cardiovascular health
  • cancer prevention

Levels of vitamin C are quickly depleted during infection. Our vitamin C is specially buffered to reduce acidity.

Vitamin C CapsulesVitamin C Buffered Capsules

One full gram of buffered vitamin C in every capsule.

An excellent source of antioxidant support, Buffered Vitamin C uses pure crystalline ascorbic acid to supply 1 gram of vitamin C in each capsule. This well-tolerated vitamin C formula supports a healthy immune system response and helps maintain healthy skin, collagen, and connective tissues.

Each (one) Capsule contains:
Vitamin C (ascorbic acid) – 1000 mg
Calcium (as calcium carbonate) – 20 mg
Magnesium (as magnesium carbonate) – 12 mg

Suggested dose: 1 capsule, 1-3 times per day, OR 1 capsule every 1-2 hours during acute illness, OR 1 capsule 3-4 times a day for accelerated vitamin C therapy.

Vitamin C Buffered Capsules – Product # 266 (60 Capsules) $15.95

Buffered Vitamin C Does Not Contain

  • artificial coloring
  • artificial flavoring
  • corn
  • dairy products
  • gluten
  • ingredients of animal origin
  • preservatives
  • salt
  • soy
  • sugar
  • wheat
  • yeast

Other Ingredients: vegetable capsule (modified cellulose), and ascorbyl palmitate.

Vitamin C Buffered Crystals

Description – High potency buffered vitamin Cpreparation in an effervescent, mineral-rich blend. Mixes easily in water or other beverages.

Each 1/4 teaspoon contains:
Vitamin C – 1066 mg
Calcium (calcium ascorbate) – 117 mg

Suggested dose: 1/4 teaspoon, 1-3 times per day, OR 1/4 teaspoon every 1-2 hours during acute illness, OR 1/4 teaspoon 3-4 times a day for accelerated vitamin C therapy.

Vitamin C Buffered Crystals – Product # 146 (8.8 ounces) $18.95

Tocotrienols


The “Other” Vitamin E

Tocotrienols“Vitamin E” is actually a mixture of tocotrienols and tocopherols, two forms of the same vitamin. Like tocopherols (the “classic” vitamin E), tocotrienols are also potent antioxidants that protect against lipid peroxidation (the damaging of fats by free radicals).

Research has shown that although tocotrienols and tocopherols possess similar activity, they work slightly differently in the body. The very newest research shows that tocotrienols may be even more valuable to good health than the classic form of vitamin E, tocopherols.

Actions of tocotrienols:

  • cholesterol-lowering properties not seen with regular vitamin E (tocopherols).
  • Tocotrienols reduce AGEs (advanced glycosylated end-products),
  • encourage healthy blood pressure levels and arterial health
  • support normal blood sugar balance
  • prevent fat oxidation

Both gamma- and delta-tocotrienols are powerful antioxidants working at the cells’ surface. Evidence indicates that tocotrienols are absorbed better than tocopherols.

The typical recommendation is 140 to 360 mg per day. Most studies have used 200 mg daily.

Contains: Tocotrienols 100 mg ( 90% Delta- Tocotrienols and 10% Gamma- Tocotrienols)

Other ingredients: Rice bran oil, gelatin, glycerin, water.

Suggested Dose: 1 or 2 softgels, one or two times daily or as directed by a healthcare practitioner.

References:

1. Kamal-Eldin A, Appelqvist LA. The chemistry and antioxidant properties of tocopherols and tocotrienols. Lipids 1996;31:671–701 [review].
2. Kamat JP, Devasagayam TPA. Tocotrienols from palm oil as potent inhibitors of lipid peroxidation and protein oxidation in rat brain mitochondria. Neurosci Lett 1995;195:179–82.
3. Sen CK, Khanna S, Roy S. Tocotrienols: Vitamin E beyond tocopherols. Life Sci. 2006;78:2088-98.
4.) Schaffer S, Muller WE, and Eckert GP. Tocotrienols: constitutional effects in aging and disease. J Nutr. 2005;135:151-4.
5.) Theriault A, Chao JT, Wang Q, et al. Tocotrienol: a review of its therapeutic potential. Clin Biochem 1999;32:309–19 [review].

SMOKING…….JUST THE FACTS

  • Smoking weakens the immune system by inhibiting cellular immunity.
  • Tobacco smoke contains carbon monoxide, a substance that is toxic to the brain.
  • Tobacco smoking is associated with a higher incidence of gingivitis and tooth loss.
  • Tobacco smoke contains cadmium, a heavy metal that can cause high blood pressure, kidney stones, and other toxic symptoms.
  • Tobacco smoke induces the formation of free radicals – highly reactive molecules that can bind to normal, healthy cells and destroy them.
  • Smokers have a higher incidence of peptic ulcer disease, a decreased response to anti-ulcer medications, and a higher mortality from peptic ulcer.
  • Female smokers are at higher risk of developing osteoporosis.
  • Female smokers are at higher risk for premature menopause.
  • Smoking accelerates skin aging and wrinkle formation.
  • Smoking causes a decrease in penile blood flow and can cause impotence in males.
  • Smokers have a three to five-fold increase in coronary artery disease compared to non-smokers.
  • Smoking is associated with the development of urinary tract cancer, bowel cancer, pancreatic cancer, cervical and uterine cancer – and yes, lung cancer.
  • Smoking is a potent risk factor for atherosclerosis.
  • 40% of heavy smokers die before they reach retirement age.
  • Nicotine causes adrenaline release, which can cause anxiety, heart palpitations, diarrhea, and high blood pressure.
  • Hydrogen cyanide, a chemical in tobacco smoke, causes inflammation of the bronchi which leads to bronchitis. Chronic obstructive pulmonary disease and emphysema often eventually result.
  • The adrenal stimulation caused by nicotine can aggravate hypoglycemia. Eventually, adrenal exhaustion results.
  • The American Lung Association reports that 350,000 Americans die each year from cigarette smoking. This is more than the combined deaths from illegal drugs, traffic accidents, suicide, homicide, and alcohol.

Don’t Kid Yourself.
Smoking tobacco is incompatible with a healthy lifestyle.

 

Sinus Infection (Sinusitis)


Natural Support For This Troublesome Condition

Sinusitis is an infection of the sinus passages, usually the frontal (head/eyes) or nasal sinuses. Symptoms may include a thick nasal discharge, pain or tenderness over the involved sinuses, headache and sometimes fever with chills. Such infection can be a one-time occurrence but is more commonly chronic and recurrent.

Recent studies have shown that antibiotic therapy is largely useless for sinusitis. Only in cases of severe pain or when symptoms have been present for more than two weeks are antibiotics sometimes indicated. Another study has shown that over 90% of people with sinusitis have some degree of yeast or fungus growing in their sinus cavities.

Anything that causes swelling of the mucous membrane (internal “skin” that lines the sinus cavities) can block normal drainage of the sinuses and lead to infection. Common causes of chronic & recurrent sinusitis include food allergy, respiratory allergy, low immune function and dental infection. (Dental infection is a frequently-overlooked cause of chronic sinusitis).

Treatment involves both acute management AND correction of underlying, predisposing factors.

Diet and Lifestyle Recommendations

  • For acute sinusitis: Follow recommendations for diet listed under Colds and Flu. Avoid the foods listed below under “chronic sinusitis” during an acute attack.
  • For chronic sinusitis: Assess for food allergies. Milk, wheat, corn, citrus and peanut butter are common food allergens associated with sinusitis. It may be wise to avoid these foods until definitive allergy-testing is completed.
  • Have a dental check-up if you have not had one in the past six months.

Primary Support

  • Maxi Multi: 3 caps, 3 times per day with meals. Optimal (not minimal) doses of antioxidant nutrients (vitamin A, beta carotene, C, E, zinc, selenium) and bioflavonoids are especially important in treating sinusitis.
  • Max EPA (Omega-3 rich fish oil): 1-2 caps, 3 times per day with meals (target dose: 3-6 caps per day). Fish oil is anti-inflammatory.
  • Immune support: 2 caps, 1-2 times per day for general immune enhancement.

Additional Support

During acute episodes:

  • Bromelain: 2 caps, 4 times per day between meals. With improvement, decrease to 1 cap, 3-4 times per day. Bromelain thins mucous so it can exit the sinus passages. It will also aid with mucous digestion once this gunk makes it’s way to the stomach.
  • Use all recommendations for acute infection.
  • Use hot packs over the sinuses during acute attacks for pain relief and decongestion.
  • Inspirol inhalant – the most potent inhalant you’ve ever used! Breathe this at least 4 times per day, but up to hourly or more if needed, during acute infection.

Tests

  • Review the symptoms of Candidiasis. If you have more than 4 of the listed symptoms, consider having a Candida test performed.

Dr. Myatt’s Comment

Correction of the underlying factors involved in chronic or recurrent sinusitis, such as food allergy and Candidiasis, will nearly always correct the problem. If you have had chronic sinusitis for many years, be patient. Complete correction may take a year or more. Patients who have overcome chronic sinusitis problems tell me it’s well worth the effort.

 

Vitamins and Mineral Supplements

Your Concise Guide To Nutritional Supplements

Definitions

Vitamins are organic compounds that are necessary for human life and health. Vitamins cannot be manufactured in the body (vitamin B12 is an exception) and so must be obtained from diet.

Minerals are inorganic ions (metals) that are also necessary for life and health. Minerals are not manufactured in the body and so must be obtained from diet.

Trace minerals are minerals necessary to the body in extremely small, or “trace,” amounts.

Accessory nutrients are substances that are not absolutely necessary for life and health (as vitamins and minerals are), but that participate with vitamins and minerals in numerous biochemical reactions.

Vitamins: What You Should be Taking, and Why

Taking vitamins is a wise health and prevention measure. Deficiencies of vitamins and minerals cause many diseases. Adding vitamins and minerals in supplemental form is an inexpensive “insurance policy” against some of the worst diseases of modern times.

A deficiency of vitamins and minerals are associated with these diseases:

A deficiency of antioxidant vitamins and minerals (especially beta carotene, vitamins C & E, and selenium) is associated with higher incidence of cancers of the colon, breast, prostate, mouth, lungs and skin. Some researchers believe that antioxidant vitamin and mineral deficiencies may be related to higher incidence of all cancers.

A mineral deficiency, especially magnesium and potassium but also calcium, is associated with high blood pressure.

Deficiencies of vitamins E, C, B6, B12, folic acid (a B vitamin), and bioflavonoids are associated with cardiovascular disease. The connection between vitamin E and heart health is so well established that conventional medical cardiologists are instructed to recommend vitamin E to their patients.

Healthy bones, and the prevention of osteoporosis, depend on sufficient levels of minerals, including calcium, magnesium, boron, zinc, copper, B vitamins, and vitamin D.

In males, benign prostatic hypertrophy is associated with decreased levels of zinc. Zinc deficiency also correlates to decreased immune function. Hypoglycemia (low blood sugar) and diabetes (high blood sugar) occur more frequently in people who are chromium deficient. After diabetes is present, low levels of vitamins A, C, E, plus zinc, selenium, choline, bioflavonoids and B complex vitamins are associated with more complications from the disease.

This list could go on for pages, but you get the idea. A deficiencyof key vitamins and mineralsare correlated with disease. Such vitamin deficiencies are also common in the modern American diet. Depleted soils result in lowered vitamin and mineral content in produce AND Americans eat less fresh produce than ever before. Much of our food is highly processed, removing not only vitamins and minerals but also fiber and enzymes.

The best health insurance may not be an expensive medical policy, but the addition of sufficient vitamins to fill in the gaps in our day-to-day nutritional status.

Some people take a wide array of individual and/or exotic supplements, but these should NOT replace a basic, healthful level of vitamin supplementation. I have listed the best and most complete formulas for basic multiple vitamin and mineral supplementation. I recommend this for all adults over age 18. If you have a special medical condition, consult an holistic physician for further recommendations. (See Telephone Consultations with Dr. Myatt)

Basic Vitamins and Minerals Supplement Program (For health maintenance in healthy individuals OR as the basis of a health program in those with known health problems). 1) Multi Vitamin / Mineral formula without iron (unless your doctor has specifically told you to take iron). There is no such thing as a good multiple vitamin supplement in a single pill. Optimal daily dosage levels of essential vitamins and minerals do not fit into one tablet or capsule. Expect to be taking 6 to 9 capsules or tablets to fulfill Optimal Daily Doses of key vitamins.

Modern Dietetics In A Nutshell

Nutritional Deficiencies

It has long been recognized that the human body will not function efficiently without vitamins and minerals. In fact, serious diseases and death result when nutrient levels become too low. Because vitamins and minerals are necessary for every chemical reaction in the body, an excess or deficiency can greatly alter physical function.

“RDA’s” (nutrient levels recommended by the U.S. Department of Agriculture) are sufficient to prevent serious deficiency-caused illnesses. (Rickets due to vitamin D deficiency, for example). They are not sufficient for optimal health and well-being.

Many scientists today agree that higher levels of certain nutrients are necessary to protect us from disease. It is also an accepted fact that even small deficiencies of nutrients can result in a decline in physical health, often before modern medicine can name a “disease.” Such deficiencies are called “subclinical,” (meaning “before they are a diagnosable illness”) and are the precursors to more serious illness.

The Standard American Diet (S.A.D.) is typically excessive in calories while being deficient in vitamins, minerals, and accessory nutrients. This is probably due to several factors: easy availability of refined-flour, high sugar foods; extensive processing of foods (which removes nutrients and fiber); and plant foods grown in mineral-deficient soils.

In addition, increased environmental exposure to toxic substances increases the body’s need for certain nutrients, especially antioxidants. (See Antioxidants.)

To ensure that you are obtaining optimal dietary nutrient levels, examine your current diet in view of the vitamin/mineral/accessory nutrient guide below. Keep a three-day diet diary to assist in calculating your baseline level of nutrient intake. Then, make dietary changes and take nutritional supplements as needed to ensure daily optimal nutrient intake.

Which Vitamin Formula is Right For You?

If you are a: Multiple Formula Antioxidants Comments Man Maxi Multi OR Once Daily MyPacks Included in Maxi Multi and MyPacks A separate antioxidant is usually needed with other multiples, not with these. Woman of Childbearing Age Nutrizyme with iron (see comment) OR Once Daily MyPacks Included in Maxi Multi and MyPacks Take a multiple WITH iron if you have heavy menstrual flow. Post-Menopausal Woman Maxi Multi OR Once Daily MyPacks Included in Maxi Multi and MyPacks Take additional Cal-Mag Amino to total 1200-1500 mg calcium per day if you are at risk for Osteoporosis. Senior Maxi Multi OR Nutrizyme with iron (see comments) Included in Maxi Multi and MyPacks Take a formula with iron only if directed to do so by your doctor. Children Children’s Multi-Vitamin and Minerals Children’s Antioxidants Specially formulated for children ages 4-12.

Vitamins

vitamin major functions major deficiency associations optimal adult dose range best food sources cautions/
notes vitamin A bone formation
skin health vision night blindness, dry eyes,
skin diseases 5,000-10,000 IU fish liver oils Do not take more than 50,000 IU per day for 3 months without medical supervision.

beta-carotene

converted to vitamin A in the body; antioxidant ulcerative colitis, skin diseases, smoking 10,000-50,000 IU green and yellow vegetables; carrots Use only natural beta-carotene; high doses may cause yellow skin (harmless).

vitamin D

increases calcium absorption;
decreases overall mortality rate osteoporosis, rheumatic pains, dental disease,
cancer,
impaired immunity 800-5,000 IU or as
directed by a physician. SUNSHINE! fish liver oil egg yolk The current daily dose of 400IU may be be set too low for optimal health.

vitamin E (tocopherol)

cellular respiration; antioxidant heart disease neurological aging 200-800 IU wheat germ oil, nuts, whole grains, egg yolk Doses over
800 IU day may elevate triglycerides.

vitamin K

blood clotting factor; bone formation osteoporosis 20-100 mcg broccoli, spinach, green tea, green cabbage, tomato Do not supplement if you are on anti-epileptic medication.

vitamin C

collagen synthesis, anti-viral, wound healing, antioxidant joint pain/arthritis, atherosclerosis, bleeding gums, decreased immunity 300-3,000 mg broccoli, red pepper, citrus fruits, cabbage At high doses, vitamin C will loosen the bowels.

vitamin B1 (thiamine)

energy processes fatigue, mental confusion, neuropathy 5-100 mg eggs, berries, nuts, legumes, liver, yeast Nontoxic.

vitamin B2 (riboflavin)

energy processes, wound healing, activates other B vitamins infection, cataracts, blurred vision, eye surgery 5-100 mg green leafy vegetables, eggs, organ meats Nontoxic. Higher doses will make urine a harmless, bright yellow.

vitamin B3 (niacin)

energy processes depression, tension headaches, memory loss 20-100 mg milk, eggs, fish, whole meal wheat flour Doses greater than 50mg may cause a skin flush. Take high doses only with doctors supervision.

vitamin B5(pantothenic acid)

energy processes; adrenal gland function allergies, morning stiffness; fatigue; muscle cramps 10-1,000 mg eggs, yeast, liver No known toxicity.

vitamin B6(pyridoxine)

energy processes; antibody formation insomnia, irritability, atherosclerosis 5-200 mg wheat germ, yeast, whole grains Oral contraceptive use increases need for this vitamin.

Folic acid

red blood cell formation, RNA/DNA synthesis fatigue, depression, atherosclerosis 200-800 mcg beans, green leafy veggies, yeast Do not take with Phenobarbital or dilantin.

vitamin B12

red blood cell formation; energy processes atherosclerosis, memory loss, GI symptoms 10-1,200 mcg fermented soy products; root veggies Nontoxic.

Biotin

energy processes; blood sugar regulation muscle pain, depression 300-600 mcg egg yolks, whole wheat No known toxicity.

Minerals

Mineral: functions deficiency associations adult dose range food sources cautions

*Calcium

bone & tooth formation; heart & muscle function osteoporosis, bone spurs, muscle cramps, rheumatism 200-1500 mg barley, kale, unrefined grains; milk, green veggies Prolonged excess may cause a mineral imbalance.

*Magnesium

energy processes, nerve function, enzyme activation stress, senility, osteoporosis, insomnia 150-600 mg avocados, almonds, whole grains, grapefruit Doses over 400 mg can cause diarrhea in some people.

Potassium

pH balance, nerve function stress, atherosclerosis, high blood pressure 1800-5625* mg * a normal diet should contain sufficient potassium potato peel, bananas, beans, almonds, whole grains Do not take high supplemental doses (food Sources are O.K.) when taking heart medicine without physician guidance.

Sodium

pH balance, nerve function Excess is more common and is assoc with high blood pressure limit daily intake to 1,500 mg okra, celery, black mission figs Very few people (athletes, diarrhea /vomiting) need to supplement.

Phosphorus

energy production, bones/teeth, B Vit. activation tooth/gum disorders, impotence, equilibrium 300-600 mg barley, beans, fish, lentils, dark green veggies Prolonged, large doses can cause calcium deficiency or mineral imbalance.

Iron

Red Blood cell production dizziness, depression, anemia 10-30 mg blackberries, cherries, spinach Do NOT take iron unless told to do so by your doctor. Iron excess is associated with health problems.

*Zinc

co-factor in numerous metabolic processes prostate enlargement, immune deficiency; atherosclerosis 15-50 mg wheat germ, wheat bran, pumpkin seed, avocado, sea food Large doses (50mg, day) can cause a copper deficiency & other mineral imbalances.

*Copper

Red blood cell production; skeletal, heart & muscle function osteoporosis, digestive function, nerve disorders 2-3 mg green leafy veggies, almonds, beans, sea food Higher doses can be toxic.

*Manganese

glandular function, bone & ligament health  diabetes, asthma, digestive disturbance 2-10 mg nuts, seeds, avocados, grapefruit, apricots High doses may create other mineral imbalances.

*Chromium

glucose metabolism; blood sugar regulation; heart function atherosclerosis, diabetes, hypoglycemia, high cholesterol, overweight 200-500 mcg whole grain cereals, molasses, meat, yeast Nontoxic at therapeutic levels.

*Selenium

antioxidant, synergistic with vitamin E cancer prevention; aging 100-200 mcg bran, whole grains, tuna, broccoli, onion Prolonged excess may be toxic. * indicates minerals most often deficient in the diet. Other minerals not marked with a * usually do not need to be supplemented. Other minerals and trace minerals include: molybdenum, flourine, chlorine, cobalt, silicon, boron, sulphur, vanadium

ACCESSORY NUTRIENTS

Bioflavonoids – compounds found in most plants in association with vitamin C. Bioflavonoids are potent antioxidants. Higher dietary levels are useful in heart disease and atherosclerosis, bleeding gums, weak immune system, inflammation, varicose veins, hayfever.

CoQ10 – (ubiquinone) A naturally-occurring compound in the human body that is a vital co-factor in energy production. Conditions benefited by increased CoQ10 levels include: cardiovascular disease, angina, congestive heart failure, mitral valve prolapse, immune deficiency, obesity, diabetes, periodontal disease, cancer, muscular dystrophy. Also use in longevity and rejuvenation programs.

Fiber – Plant cell walls present in whole grains, legumes, fruits and vegetables. This part of the plant is usually lost in processing. Fiber deficiency is associated with numerous illnesses: obesity, atherosclerosis, diabetes, gallstones, varicose veins, constipation, diverticulosis, irritable bowel, colon cancer, high blood pressure and high cholesterol.

FOS (fructooligosaccharides) Naturally- occurring sugar-like substances that act as food to friendly GI bacteria. In human body cells, this substance is not utilized as energy (or as a true sugar), but to probiotic gut bacteria, FOS is a banquet. The addition of FOS to probiotic formulas (as in Enterogenic concentrate, product # 218), helps good bacteria re-colonize the GI tract faster and more plentifully.

Friendly bacteria – (probiotics) The naturally-occurring bacteria of the colon help protect us from many conditions, including candidiasis, allergies, constipation, B12 vitamin deficiency. These good bacteria are damaged or destroyed by dietary imbalances, antibiotic and other drug use. Replacement of good bacteria results in improved colon function.

Glucosamine sulfate – A naturally occurring substance that has been found to be highly effective in treating osteoarthritis. It acts both to reduce pain and to stimulate joint repair.

5-Hydroxy-Tryptophan-(5-HTP)
5-HTP is the intermediate metabolite of the amino acid L-tryptophan. This amino acid intermediate participates in the body’s production of serotonin. It also stimulates increased endorphin, melatonin, norepinephrine and dopamine production. These brain chemicals (neuro-transmitters) help increase energy, improve mood and sleep, and decrease appetite. Useful for insomnia, mood disorder (anxiety/depression) and weight loss programs.

L-Carnitine – an amino acid that is crucial to normal energy production and fat metabolism. Carnitine has been shown to benefit atherosclerotic heart disease and high cholesterol and triglycerides. Improves fat metabolism throughout the body.

L-Glutathione – A tri peptide (3 amino acids) that acts as a potent antioxidant in the body. Supplementation is useful in allergies, cancer prevention, liver detoxification, cataracts, heavy metal toxicity, longevity and rejuvenation.

Omega-3 Oils are derived from fatty fish and flax seeds. These fatty acids are anti-inflammatory and have a positive effect on cardiovascular disease, including high cholesterol and high blood pressure, allergic and inflammatory conditions (including psoriasis and eczema), autoimmune diseases, cancer, neurological disease, menopause, general health enhancement.

Omega-6 Oils found in evening primrose, black currant, borage and a number of vegetable oils. Although supplementation is popular, these oils increase arachadonic acid levels (an inflammatory substance). Only diabetics need to supplement very small doses of this oil. (less than 500mg/day).

Pau ‘d’ Arco


(Tabebuia spp.) [a.k.a. Lapacho]

Actions: Anti-tumor; anti-Candidal; antibiotic; immune stimulant; anti-inflammatory; tonic.

Uses: Candidiasis; fungus; immune stimulation; infections; cancer.

What’s Burning You?

The REAL Cause of Heartburn, Indigestion and GERD and “Sour Stomach”

Older people have considerably more digestive problems than younger folks, and this has typically but incorrectly been blamed on over-production of stomach acid. Not only have medical studies debunked excess stomach acid as the cause of indigestion, but common sense debunks the myth as well.

Why does this matter? Because the chronic use of antacids and acid-blocking drugs for indigestion has some dangerous and even deadly side-effects

The “Acid Over-Production” Myth Debunked

Do you really think that some bodily function starts working better with age? Hahahaha!

With age, nothing works as well as it did in earlier years. I hope I’m not popping anyone’s bubble here.

Come on – we don’t move as fast at age 57 as we did at 27. Vision and hearing are typically less acute in our 70s than they were in our 30s. Skin is less elastic at 69 than at 29. Production of hormones and body fluids decreases with age. Why would we think that our stomachs do the opposite of all other organs and become more active with age instead of less active? Only a drug salesman or a pill-pushing doctor would try to convince us of such foolishness.

The stomach’s primary job is to digest protein and emulsify fats, and it does this by making an extremely powerful acid called hydrochloric acid (HCL) and a protein-digesting enzyme called pepsin. The hydrochloric acid made by a healthy stomach is one million times stronger than the mild acidity of urine or saliva. A leather-like strip of jerky can be quickly turned into “beef soup” by the action of hydrochloric acid and pepsin in the stomach. That’s how normal digestion is supposed to work.

But just like the rest of an aging body, the stomach’s hydrochloric acid and pepsin production decreases over time. As a result, we do not digest food as well. The term “indigestion” implies lack of digestion, not over-digestion. This is why we can’t eat a whole pepperoni pizza washed down with a bottle of soda like we did when we were teenagers. Our aging stomachs don’t have the same digestive vigor – strong hydrochloric acid and pepsin – to digest food like youthful stomachs do.

Medical Science Verifies Low Acid Production

OK, that’s the common sense of it. Now here’s the science. Many older studies conducted on several thousand people in the 1930?s and 1940?s showed that half of all people by age 60 were functioning at only 50% gastric acid output. Numerous contemporary studies verify that that stomach acid production often declines with age.

The Bottom Line: when someone over age 40 has chronic or chronic / intermittent indigestion, that indigestion is almost certainly due to a weaker stomach with less acid and pepsin output, not a stronger stomach making more digestive juices.

“But My Symptoms Feel Like Too Much Acid…”

Strong stomach acid and pepsin quickly “emulsify” fats and proteins, making them ready for the next step of digestion, passage into the small intestine. When these digestive factors are weak, food remains in the stomach for longer and it begins to ferment. Gas pressure from the fermentation can cause bloating and discomfort and can can also cause the esophageal sphincter to open, allowing stomach contents to “backwash” into the esophagus.

Even though weak stomach acid is the central cause of this, even this weak stomach acid, which has no place in the esophagus, will “burn.” This burning sensation confuses many people, including doctors, who then “ASSuME” that excess acid is to blame. Too little acid, resulting in slowed digestion, and gas which creates back-pressure into the esophagus is the real cause of almost all “heartburn” and GERD.

Why People Take Acid-Blockers

Why in the world would anyone take antacids or acid blockers to correct a deficiency of stomach acid? In two words: symptom relief.

But if heartburn or gastro esophageal reflux disease (GERD) are caused by too little stomach acid, why does blocking more of the acid relieve the discomfort? And why isn’t that a good thing to do?

Remember, even weak stomach acid does not belong in the esophagus. When ALL acid production is blocked, the “backwash” of stomach contents into the esophagus will not burn. However, repeatedly using this “band-aid” method has some serious long-term consequences.

The Dangers of Antacids and Acid-Blocking Drugs

Our bodies need 60 or so essential nutrients. “Essential” means that the body MUST have this nutrient or death will eventually ensue, and the nutrient must be obtained from diet because the body cannot manufacture it. Many of these essential nutrients require stomach acid for their assimilation. When stomach acid production declines, nutrient deficiencies begin.

Calcium, for example, requires vigorous stomach acid in order to be assimilated. Interestingly, the rate of hip replacement surgery is much higher in people who routinely use antacids and acid-blocking drugs. We know that people who have “acid stomach” were already having trouble assimilating calcium from food and nutritional supplements due to lack of normal stomach acid production. When these symptoms are “band-aided” with drugs which decrease stomach acid even more, calcium assimilation can come to a near-halt. The result? Weak bones, hip fractures and joint complaints resulting in major surgery.

Jonathan Wright, M.D., well-known and respected holistic physician, states that:

“Although research in this area is entirely inadequate, its been my clinical observation that calcium, magnesium, iron, zinc, copper, chromium, selenium, manganese, vanadium, molybdenum, cobalt, and many other micro-trace elements are not nearly as well-absorbed in those with poor stomach acid as they are in those whose acid levels are normal. When we test plasma amino acid levels for those with poor stomach function, we frequently find lower than usual levels of one or more of the eight essential amino acids: isoleucine, leucine, lysine, methionine, phenylalanine, threonine, tryptophan, and valine. Often there are functional insufficiencies of folic acid and/or vitamin B12.”

Remember, these are essential nutrients. Deficiencies of any single one of them can cause serious health problems over time. Weak bones, diminish immune function, failing memory, loss of eyesight and many other “diseases of aging” are often the result of decreased stomach function.

Ulcers can even be caused by too little acid. Surprised? We know today that most ulcers are caused by a bacterium called h. pylori. This little beastie is killed by strong stomach acid. But when stomach acid is weak, watch out! Weak stomach acid is how h. pylori gets a foot-hold. (People with active ulcers should not supplement hydrochloric acid until the ulcer has healed).

Diseases Associated with Low Gastric Function

Low stomach acid is associated with the following conditions:

  • Acne rosacea
  • Addison’s disease
  • Allergic reactions
  • Candidiasis (chronic)
  • Cardiac arrhythmias
  • Celiac disease
  • Childhood asthma
  • Chronic autoimmune hepatitis
  • Chronic cough
  • Dermatitis herpeteformis
  • Diabetes (type I)
  • Eczema
  • Gallbladder disease
  • GERD
  • Graves disease (hyperthyroid)
  • Iron deficiency anemia
  • Laryngitis (chronic)
  • Lupus erythromatosis
  • Macular degeneration
  • Multiple sclerosis
  • Muscle Cramps
  • Myasthenia gravis
  • Mycobacterium avium complex (MAC)
  • Osteoporosis
  • Pernicious anemia
  • Polymyalgia rheumatica
  • Reynaud’s syndrome
  • Rheumatoid arthritis
  • Scleroderma
  • Sjogren’s syndrome
  • Stomach cancer
  • Ulcerative colitis
  • Vitiligo

It also appears that many cases of depression, which appear related to too little neurotransmitters (which in turn are made from amino acids) may in fact be inability to absorb the necessary precursors due to – you guessed it – low stomach acid. I suspect there are a large number of other diseases that begin with a failing digestive system and that have not yet been recognized as such.

Even so, many people who have low stomach acid do not have symptoms of heartburn, “acid indigestion” or GERD.

The Gastric Acid Function Test

Here’s a simple question. Before your doctor diagnosed GERD from “too much stomach acid,” did he/she perform a stomach acid function test?

X-rays and gastroscopy do not evaluate stomach acid production. The medical test for stomach acid, called the Heidelberg test, requires swallowing a small capsule and then having it pulled back up on a “string.” You’d remember if you had this done. Interestingly, this test is ALMOST NEVER PERFORMED before excess stomach acid is diagnosed, hence the incorrect diagnosis!

Why The Blind Spot In Medicine?

From the 1800’s up until the 1950’s, hydrochloric acid (HCl) supplements (both with and without pepsin) were widely prescribed and used. Physicians simply considered replacement of digestive acid to be like replacement of thyroid hormone for a failing thyroid or hormone replacement for aging ovaries.

In the 1950’s, some badly designed and misinterpreted “research” was used to convince physicians that HCl and pepsin replacement therapy is unnecessary. Besides, the “replacement” therapy – HCL and pepsin – are natural substances that are difficult to patent. Instead, drug companies focused on patentable drugs to treat “hyperchlorhydria” (excess stomach acid), and the highly profitable prescription and OTC acid blocking drug industry was born.

Once again I ask: if a doctor diagnosed you with excess stomach acid, did he or she actually perform the Heidelberg test? If you diagnosed yourself, did you perform a gastric acid self-test? No? I rest my case.

The Gastric Acid Function Self-Test

Fortunately, the Heidelberg test is not required to arrive at a correct diagnosis of too little stomach acid. You can perform a gastric acid self-test at home using some betain HCL capsules taken with meals. If digestion improves – bingo! You’re hydrochloric acid deficient.

This issue of low stomach acid is central to so many diseases that I recommend a gastric acid self-test to EVERYONE over age 50 and anyone under age 50 who has any medical complaint related to nutrient deficiency.

I’ve put together an inexpensive yet highly effective “Gastric Acid Function Self Test Kit” that includes full instructions for testing your own stomach acid (it’s easy with the instructions) plus “test sizes” of the supplements – including hydrochloric acid and pepsin – needed for the test.

Testing your own digestive function is simple and easy, and it could save you much grief, sickness, and yes, heartburn.

References

1.) Gastric observations in achlorhydria. J Dig Dis. 1941, 8: 401-407.
2.) Gastrointestinal Tract Disorders in the Elderly, pp. 62-69. Edinburgh: Churchill Livingstone: 1984.
3.) Age related changes in gut physiology and nutritional status. Gut. 1996 Mar; 38(3):306-9.
4.) A retrospective study of the usefulness of acid secretory testing. Aliment Pharmacol Ther. 2000 Jan;14(1):103-11.
5.) Age related changes in gut physiology and nutritional status. Gut. 1996 Mar;38(3):306-9.
6.) Hypochlorhydria: a factor in nutrition. Annu Rev Nutr. 1989;9:271-85.
7.) Gastric hypochlorhydria and achlorhydria in older adults. JAMA. 1997 Nov 26;278(20):1659-60.
8.) The aging gut. Nutritional issues. Int J Nurs Pract. 2006 Apr;12(2):110-8. Summary: Aging is associated with decreased gastric output.
9.) The aging gut. Nutritional issues. Gastroenterol Clin North Am. 1998 Jun;27(2):309-24.
10.) Changes in gastrointestinal function attributed to aging. Am J Clin Nutr. 1992 Jun;55(6 Suppl):1203S-1207S.
11.) Digestive function and aging. Hum Nutr Clin Nutr. 1983 Mar;37(2):75-89.
12.) Symptomatic gastro-oesophageal reflux in a patient with achlorhydria. Gut. 2006 Jul;55(7):1054-5.
13.) Effects of aging process on digestive functions. Compr Ther. 1991 Aug;17(8):46-52.
14.) Fundic atrophic gastritis in an elderly population. Effect on hemoglobin and several serum nutritional indicators. J Am Geriatr Soc. 1986 Nov;34(11):800-6.
15.) Vitamin B12 (cobalamin) deficiency in elderly patients. CMAJ. 2004 Aug 3;171(3):251-9.
16.) Anemia caused by vitamin B 12 deficiency in subjects aged over 75 years: new hypotheses. A study of 20 cases. Rev Med Interne. 2000 Nov;21(11):946-54.
17.) Cobalamin, the stomach, and aging. Am J Clin Nutr. 1997 Oct;66(4):750-9.
18.) Age-related changes in cobalamin (vitamin B12) handling. Implications for therapy. Drugs Aging. 1998 Apr;12(4):277-92.
19.) Intestinal malabsorption in the elderly. Digestive Diseases. 2007;25(2):144-50.
20.) Gastric acid secretion in chronic iron-deficiency anaemia. Lancet. 1966 Jul 23;2(7456):190-2.
21.) Involvement of the corporal mucosa and related changes in gastric acid secretion characterize patients with iron deficiency anaemia associated with Helicobacter pylori infection. Aliment Pharmacol Ther. 2001 Nov;15(11):1753-61.
22.) The aging process as a modifier of metabolism. Am J Clin Nutr. 2000 Aug;72(2 Suppl):529S-32S.
23.) Low gastric hydrochloric acid secretion and mineral bioavailability. Adv Exp Med Biol. 1989;249:173-84.
24.) Effects of pH on mineral-phytate, protein-mineral-phytate, and mineral-fiber interactions. Possible consequences of atrophic gastritis on mineral bioavailability from high-fiber foods. J Am Coll Nutr. 1988 Dec;7(6):499-508.
25.) Long-term proton pump inhibitor therapy and risk of hip fracture. JAMA. 2006 Dec 27;296(24):2947-53.
26.) Antral atrophy, Helicobacter pylori colonization, and gastric pH. Am J Clin Pathol. 1996 Jan;105(1):96-101.
27.) High acid secretion may protect the gastric mucosa from injury caused by ammonia produced by Helicobacter pylori in duodenal ulcer patients. J Gastroenterol Hepatol. 1996 Jul;11(7):674-80.
28.) Rosacea keratitis and conditions with vascularization of the cornea treated with riboflavin. Arch Ophthamol 1940;23:899–907.
29.) Incidence of anti-Helicobacter pylori and anti-CagA antibodies in rosacea patients. Int J Dermatol. 2003 Aug;42(8):601-4.30.) Gastrointestinal findings in atopic children. Eur J Pediatr 1980;134:249–54.
31.) Suppression of gastric H2-receptor mediated function in patients with bronchial asthma and ragweed allergy.
Chest 1986;89:491–6.
32.) Allison JR. The relation of hydrochloric acid and vitamin B complex deficiency in certain sk
in diseases. South Med J 1945;38:235–41.
33.) Effect of hydrochloric acid on iron absorption. N Engl J Med 1968;279:672–4.
34.) The importance of gastric hydrochloric acid in the absorption of nonheme food iron. J Lab Clin Med 1978;92:108–16.
35.) Bray GW. The hypochlorhydria of asthma in childhood. Q J Med 1931;24:181–97.
36.) Candida overgrowth in gastric juice of peptic ulcer subjects on short- and long-term treatment with H2-receptor antagonists. Digestion.1983;28:158–63.
37.) Antibacterial activity of the pancreatic fluid. Gastroenterology 1985;88:927–32 [review].
38.) Non-immunological defense mechanisms of the gut. Gut 1990;33:1331–7 [review].
39.) Characterization of gastric mucosal lesions in patients with celiac disease: a prospective controlled study.Am J Gastroenterol. 1999 May;94(5):1313-9.
40.) Chronic cough due to gastroesophageal reflux disease: failure to resolve despite total/near-total elimination of esophageal acid. Chest. 2002 Apr;121(4):1132-40.
41.) Gastric lesion in dermatitis herpetiformis.Gut.1976 Mar;17(3):185-8.
42.) Auto-immune atrophic gastritis in patient with dermatitis herpetiformis. Acta Derm Venereol. 1976;56(2):111-3.
43.) Predictive value of gastric parietal cell autoantibodies as a marker for gastric and hematologic abnormalities associated with insulin-dependent diabetes. Diabetes. 1982 Dec;31(12):1051-5.
44.) Parietal cell antibodies and gastric secretion in children with diabetes mellitus. Acta Paediatr Scand. 1980 Jul;69(4):485-9.
45.) Oesophageal acid exposure and altered neurocardiac function in patients with GERD and idiopathic cardiac dysrhythmias. Aliment Pharmacol Ther. 2006 Jul 15;24(2):361-70.
46.) Capper WM, Butler TJ, Kilby JO, Gibson MJ. Gallstones, gastric secretion and flatulent dyspepsia. Lancet 1967;i:413–5.
47.) Gastric juice nitrite and vitamin C in patients with gastric cancer and atrophic gastritis: is low acidity solely responsible for cancer risk? Eur J Gastroenterol Hepatol. 2003 Sep;15(9):987-93.
48.) Correlation of ratio of serum pepsinogen I and II with prevalence of gastric cancer and adenoma in Japanese subjects. Am J Gastroenterol. 1998 Jul;93(7):1090-6.
49.) Atrophic body gastritis in patients with autoimmune thyroid disease: an underdiagnosed association. Arch Intern Med. 1999 Aug 9-23;159(15):1726-30.
50.) Early manifestations of gastric autoimmunity in patients with juvenile autoimmune thyroid diseases.J Clin Endocrinol Metab. 2004 Oct;89(10):4944-8.
51.) Review article: the role of pH monitoring in extraoesophageal gastro-oesophageal reflux disease. Aliment Pharmacol Ther. 2006 Mar; 23 Suppl 1:40-9. Summary: association with laryngitis, non-cardiac chest pain, etc.
52.) Age-Related Eye Disease Study Group. Risk factors associated with age-related macular degeneration. Opthamology.
53.) Altered gastric acidity in patients with multiple sclerosis. Cesk Gastroenterol Vyz. 1968 Dec;22(8):526-30.
54.) Gastroesophageal reflux disease, acid suppression, and Mycobacterium avium complex pulmonary disease. Chest. 2007 Apr;131(4):1166-72.
55.) Malabsorption of vitamin B12 in dermatitis herpetiformis and its association with pernicious anaemia. Acta Med Scand. 1986;220(3):261-8
56.) Small intestinal bacterial overgrowth in patients with rheumatoid arthritis. Ann Rheum Dis. 1993 Jul;52(7):503-10.
57.) Hartung EF, Steinbroker O. Gastric acidity in chronic arthritis. Ann Intern Med 1935;9:252.
58.) Hypochlorhydria and hypergastrinaemia in rheumatoid arthritis. Ann Rheum Dis. 1979 Feb;38(1):14-7
59.) Francis HW. Achlorhydria as an etiological factor in vitiligo, with report of four cases. Nebraska State Med J 1931;16(1):25–6.

Vitamin D A Special HealthBeat News Report


Vitamin D – You have been reading about it in the news, and you have wondered what is real and what is hype.

Dr. Myatt and Nurse Mark have researched and prepared this special report for HealthBeat News Readers.

Vitamin D — The Short Course

1.) Vit D is produced in our bodies in response to sun exposure. Vit D is also available from food and supplements.

2.) Vit D is FAR more important to health than was previously realized. I’m talking FAR more important.

3.) Vit D deficiency is widespread, including North America, even in sunny climates like Arizona. Many people who think they are getting enough Vitamin D from sunlight are mistaken.

4.) How to Optimize Vit D Levels for Good Health:

I.)  Vit D test, supplement accordingly, re-test

II.) Supplement at 5,000IU for 3 months, then test your levels.

III.) Don’t test, run the risk of being deficient, but take at least 2,000IU total per day. (This is still an extremely conservative dose, but much higher than the RDA of 400IU which hasn’t been changed yet to reflect the newer findings about Vit D). 

5.) Natural ways to obtain Vit D: Foods, supplements and sun exposure.

Vitamin D — Nutrient of the Decade: Are You Getting Enough?

The Consequences of Low Vitamin D

Vitamin D is called “the sunshine Vitamin” because our bodies make it in response to sun exposure.

Vit D is necessary for normal bone formation in both children and adults. In children, deficiencies of Vit D lead to rickets. In adults, deficiencies are associated with osteoporosis and osteomalacia (soft bones), decreased muscle strength and increased risk of fall. (1,12,14,22,43-48)Until recently, the bone-protecting effect was  about all that Vit D was known for, but the past decade of medical research has changed all that.

The newly appreciated Vitamin D deficiency risks include:

1.) heart disease: myocardial infarction, high blood pressure, heart failure, myopathy, sudden cardiac death, stroke (11,13-26, 30, 49-50)

2.) blood sugar problems: glucose intolerance, diabetes mellitus, metabolic syndrome (13-14,19,23-24,27-29)

3.) cancer prevention and improved cancer survival rates (7,8,11,14,15,24,31-37)

4.) upper respiratory tract infections, influenza and tuberculosis (24,30,38)

5.) cognitive impairment and low mood (38-40)

6.) autoimmune disease (multiple sclerosis, RA, systemic lupus erythromatosis (SLE) (15,24,26,29,30,32,41,42)

7.) misc. diseases: psoriasis, polycystic ovarian syndrome, inflammatory bowel disease

8.) urinary incontinence (54)

9.) and all-cause mortality! (5,6,7,24,30,51)

How “significant” are these associations? Here are some of the conclusions of various studies and meta-analyses (lots of studies looked at together) concerning Vit D. Italics are mine for emphasis.

“Research strongly supports the view … Vitamin D status would have significant protective effects against the development of cancer …. cancers of the breast, colon, prostate, ovary, lungs, and pancreas…” (8)

“High levels of Vitamin D among middle-age and elderly populations are associated with a substantial decrease in cardiovascular disease, type 2 diabetes and metabolic syndrome.” (9)

“Low levels of [Vitamin D] are independently predictive for fatal strokes” (10)

“It is estimated that there is a 30 to 50% reduction in risk for developing colorectal, breast, and prostate cancer by either increasing Vitamin D intake or increasing sun exposure…” (11)

“Oral Vitamin D supplementation between 700 to 800 IU/d appears to reduce the risk of hip and any nonvertebral fractures in ambulatory or institutionalized elderly persons” (12)

” 28 studies including 99,745 participants … highest levels of serum [Vit D] were associated with a 43% reduction in cardiometabolic disorders (cardiovascular disease, diabetes and metabolic syndrome) …” (9)

Are Your Vitamin D Levels Optimal? (Vitamin D Deficiency is Widespread)

One billion people worldwide are estimated to be Vit D deficient, and the problem affects us here in the United States as well. (2) One study found that more than half of North American women receiving drugs for prevention or treatment of osteoporosis were Vitamin D deficient. (1) Another study found 48% of pre-adolescent girls to be Vit D deficient (3). Other studies have found that 40% to 100% of older men and women in both the United States and Europe are Vitamin D deficient.[2] Because of the importance of Vit D and how widespread Vit D deficiency is, an estimated $100 to $200 billion is spent (wasted) each year on diseases which may really just be Vitamin D deficiencies. [4]

Age, overweight, dark skin color, use of sunscreen, and overprotection from the sun’s rays are causes of decreased production of Vit D in response to sunlight. (52,52)

How Much Vitamin D Should You Take?

Ideally, you should take whatever amount of Vitamin D puts you in the “optimal” range. Since the amount will be highly variable depending on age, sex, race, weight, daily sun exposure and diet, there is no “one size fits all” answer. Instead, blood testing of Vitamin D levels and increasing intake until optimal levels are reached is the surest way to obtain optimal concentrations of Vitamin D in the body.

Deficiency Insufficiency Sufficiency * Optimal Excess (Toxicity) <20ng/ml 20-32ng/ml 32-100ng/ml 40-80ng/ml > 150ng/ml

* – conventional medicine says that 30 ng/ml is “sufficient.” Chart references (59-62)

At the wellness Club we believe the most accurate and effective way to embark on a program of Vit D supplementation is to perform a Vit D test, supplement Vit D in accordance with the results, and then re-test in 3 months at which time your daily doses of Vit D can be fine-tuned for maintenance. March (right now!) is the best time to test initially because Vit. D stores tend to be lowest in this month.

The Vitamin D Council, a non-profit group dedicated to Vitamin D research and education recommends people take 5,000 IU per day for 2-3 months, then perform a Vitamin D test. They then suggest adjusting the dosage so that blood levels are between 50-80 ng/mL (or 125-200 nM/L) year-round. (55)

Alternately, some people opt to supplement without knowing their initial Vit D levels. A dose of 2000IU is quite conservative but certainly safe for almost anyone. In cases of significant Vit D deficiency conservative dosing such as this may take considerable time to rebuild healthy stores of this important Vitamin.

For those who wish to calculate their own Vit D requirements, 100 IU of Vitamin D could be expected to raise blood level of 25(OH)D by 1 ng/ml. (11)

Can too much Vitamin D can be toxic? Research shows that massive doses may eventually cause toxicity. One source found that in adults a sustained intake of 50,000 IU daily could produce toxicity within a few months (58) and 40,000 IU per day in infants has been shown to produce toxicity within 1 to 4 months. (56) That is ten times the recommended dose for each of those age groups! Vitamin D testing is good insurance that will allow you to safely fine-tune your dosage to your actual needs. Be careful though, since not all testing is the same and lab references and standards vary – be sure that you are comparing “apples to apples” and obtaining useable results when you are tested.

The 25-hydroxyVitamin D blood test (25(OH)D blood test) is a test that measures the amount of calcidiol circulating in the blood. This is the most accurate measure of the amount of Vitamin D in the body. The Wellness Club offers Vitamin D testing – performed by a lab that adheres to standardized references and values so that you know what you are getting when you receive your results. This can is performed at home with a “spot” (finger stick) blood test. Other tests that require a blood draw are also available.

How to Get to Your Optimal Vitamin D Levels

Start Vitamin D supplementation eight to twelve weeks before testing. Dr. John Cannell of the Vitamin D Council suggests a starting dose of 1,000 IU per 25 pounds of body weight. For example, a 150 pound person would take 6,000 IU Vitamin D per day. (150 divided by 25 = 6; 1,000IU x 6 = 6,000). Maintain this dose for 8-12 weeks, then test.

This dose may or may not put you in the optimal target range, but it certainly won’t put you in any “toxic” range. Remember, most adults can safely take up to 10,000IU per day and still be far away from Vitamin D toxicity which typically appears at 40,000-50,000IU taken for several months.

Although this dose should theoretically put you in an optimal range, numerous personal variations alter Vitamin D requirements. Some people will need a higher dose than this calculation affords. However, taking the calculated dose should at least put you “in the ballpark” for optimal dosing.

When you test results come back, you can use the number to help you know whether or not you need to increase your Vit D dose and by how much. It is estimated that each 1,000 IU increase in supplemental Vitamin D will generally produce a 10 ng/ml increase in the Vitamin D blood level (8). If your test result shows that you are 10ng/ml below your target, increase daily Vit D intake by 1,000IU per day for a total of 7,000IU per day from the above example. Continue this dose and re-test in another 3 months to verify that you are now in your optimal range.

Congratulations! You have found your optimal daily Vitamin D intake needed to maintain optimal Vitamin D blood levels.

How to Obtain Vitamin D Naturally

Exposure to sun is the most natural way to boost Vit D levels. Medical scientists have found that the skin produces approximately 10,000 IU of Vitamin D in response to as little as 30 minutes of unprotected summer sun exposure. (57)

Vitamin D can be obtained from food too. Since rickets in children is such a crippling but preventable condition, governments have long encouraged the “fortification” of dairy products and breads and cereals with token amounts of Vitamin D. In the United States and Canada, for example, fortified milk typically provides 100 IU per glass.

It is difficult to obtain optimal levels of Vitamin D from food alone.

Food IUs per serving* Percent DV** Cod liver oil, 1 tablespoon 1,360 340 Salmon (sockeye), cooked, 3 ounces 794 199 Mushrooms that have been exposed to ultraviolet light to increase vitamin D, 3 ounces (not yet commonly available) 400 100 Mackerel, cooked, 3 ounces 388 97 Tuna fish, canned in water, drained, 3 ounces 154 39 Milk, nonfat, reduced fat, and whole, vitamin D-fortified, 1 cup 115-124 29-31 Orange juice fortified with vitamin D, 1 cup (check product labels, as amount of added vitamin D varies) 100 25 Yogurt, fortified with 20% of the DV for vitamin D, 6 ounces (more heavily fortified yogurts provide more of the DV) 80 20 Margarine, fortified, 1 tablespoon 60 15 Sardines, canned in oil, drained, 2 sardines 46 12 Liver, beef, cooked, 3.5 ounces 46 12 Ready-to-eat cereal, fortified with 10% of the DV for vitamin D, 0.75-1 cup (more heavily fortified cereals might provide more of the DV) 40 10 Egg, 1 whole (vitamin D is found in yolk) 25 6 Cheese, Swiss, 1 ounce 6 2 *IUs = International Units.

**DV = Daily Value. DVs were developed by the U.S. Food and Drug Administration to help consumers compare the nutrient contents of products within the context of a total diet. The DV for vitamin D is 400 IU for adults and children age 4 and older. Food labels, however, are not required to list vitamin D content unless a food has been fortified with this nutrient.

Table courtesy of the U.S. Government National Institutes of Health Office of Dietary Supplements

Although cod liver oil is high in Vitamin D, it is also high in Vitamin A which interferes with Vit D uptake, so cod liver oil is not the best supplemental form of Vit D. Keep daily intake of pre-formed Vitamin A to a maximum of 5,000IU per day so as not to interfere with Vitamin D absorption. Beta carotene does not appear to interfere with Vit. D uptake.

Vegetarians need to be sure they are getting plenty of sunshine, because other than tiny amounts that may be found in UV-irradiated mushrooms, there are no vegetable sources of Vitamin D.

The Bottom Line on Vitamin D

Achieving Optimal Vitamin D  levels appears to be one of the most important things we can do for our overall health and life expectancy.

Please click on the image below enjoy an interesting and instructive video which discusses the relationship between Vitamin D and Cancer.

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References

1.) Holick MF, Siris ES, Binkley N, et al. Prevalence of Vitamin D inadequacy among postmenopausal North American women receiving osteoporosis therapy. J Clin Endocrinol Metab. 2005;90: 3215-3224.
2.) Holick MF. Vitamin D deficiency. N Engl J Med. 2007;357:266-281.
3.) Sullivan SS, Rosen CJ, Halteman WA, Chen TC, Holick MF. Adolescent girls in Maine at risk for Vitamin D insufficiency. J Am Diet Assoc. 2005;105:971-974.
4.) GrassrootsHealth. The Vitamin D deficiency epidemic. A call to D*action. http://www.grassrootshealth.org/daction/epidemic.php. Accessed May 8, 2009.
5.) GrassrootsHealth. Disease incidence prevention by serum 25(OH)D level. http://www.grassrootshealth.org/_download/disease_incidence_prev_chart_101608.pdf. Accessed May 8, 2009.
6.) Autier P, Gandini S. Vitamin D supplementation and total mortality. Arch Intern Med. 2007;167(16):1730-1737.
7.) Thomas L. Lenz. Vitamin D Supplementation and Cancer Prevention. Am J Lifestyle Med. 2009;3(5):365-368.
8.) Ingraham BA, Bragdon B, Nohe A. Molecular basis of the potential of Vitamin D to prevent cancer. Curr Med Res Opin. 2008 Jan;24(1):139-49.
9.) Parker J, Hashmi O, Dutton D, Mavrodaris A, Stranges S, Kandala NB, Clarke A, Franco OH. Levels of Vitamin D and cardiometabolic disorders: systematic review and meta-analysis. Maturitas. 2010 Mar;65(3):225-36. Epub 2009 Dec 23.
10.) Pilz S, Dobnig H, Fischer JE, Wellnitz B, Seelhorst U, Boehm BO, März W. Low Vitamin d levels predict stroke in patients referred to coronary angiography. Stroke. 2008 Sep;39(9):2611-3. Epub 2008 Jul 17.
11.) Holick MF. Vitamin D and sunlight: strategies for cancer prevention and other health benefits. Clin J Am Soc Nephrol. 2008 Sep;3(5):1548-54. Epub 2008 Jun 11.
12.) Bischoff-Ferrari HA, Willett WC, Wong JB, Giovannucci E, Dietrich T, Dawson-Hughes B. Fracture prevention with Vitamin D supplementation: a meta-analysis of randomized controlled trials. JAMA. 2005 May 11;293(18):2257-64.
13.) Anagnostis P, Athyros VG, Adamidou F, Florentin M, Karagiannis A. Vitamin D and Cardiovascular Disease: A Novel Agent for Reducing Cardiovascular Risk ? Curr Vasc Pharmacol. 2010 Feb 25. [Epub ahead of print]
14.) Holick MF. Vitamin D: importance in the prevention of cancers, type 1 diabetes, heart disease, and osteoporosis. Am J Clin Nutr. 2004 Mar;79(3):362-71.
15.) Holick MF. Vitamin D and sunlight: strategies for cancer prevention and other health benefits. Clin J Am Soc Nephrol. 2008 Sep;3(5):1548-54. Epub 2008 Jun 11.
16.) Judd SE, Tangpricha V. Vitamin D deficiency and risk for cardiovascular disease. Am J Med Sci. 2009 Jul;338(1):40-4.
17.) Kendrick J, Targher G, Smits G, Chonchol M.25-HydroxyVitamin D deficiency is independently associated with cardiovascular disease in the Third National Health and Nutrition Examination Survey. Atherosclerosis. 2009 Jul;205(1):255-60. Epub 2008 Nov 11.
18.) Lee W, Kang PM. Vitamin D deficiency and cardiovascular disease: Is there a role for Vitamin D therapy in heart failure? Curr Opin Investig Drugs. 2010 Mar;11(3):309-14.
19.) Martins D, Wolf M, Pan D, Zadshir A, Tareen N, Thadhani R, Felsenfeld A, Levine B, Mehrotra R, Norris K. Prevalence of cardiovascular risk factors and the serum levels of 25-hydroxyVitamin D in the United States: data from the Third National Health and Nutrition Examination Survey. Arch Intern Med. 2007 Jun 11;167(11):1159-65.
20.) McConnell JP, Foley KF, Vargas GM. HypoVitaminosis D: a new risk marker for cardiovascular disease. Clin Lab Sci. 2009 Fall;22(4):240-6.
21.) Mertens PR, Müller R. Vitamin D and cardiovascular risk. Int Urol Nephrol. 2009 Dec 29. [Epub ahead of print]
22.) Murlikiewicz K, Zawiasa A, Nowicki M. Vitamin D–a panacea in nephrology and beyond] Pol Merkur Lekarski. 2009 Nov;27(161):437-41.{article in Polish]
23.) Parker J, Hashmi O, Dutton D, Mavrodaris A, Stranges S, Kandala NB, Clarke A, Franco OH. Levels of Vitamin D and cardiometabolic disorders: systematic review and meta-analysis. Maturitas. 2010 Mar;65(3):225-36. Epub 2009 Dec 23.
24.) Pilz S, Dobnig H, Nijpels G, Heine RJ, Stehouwer CD, Snijder MB, van Dam RM, Dekker JM. Vitamin D and mortality in older men and women. Clin Endocrinol (Oxf). 2009 Nov;71(5):666-72. Epub 2009 Feb 18.
25.) Pilz S, März W, Wellnitz B, Seelhorst U, Fahrleitner-Pammer A, Dimai HP, Boehm BO, Dobnig H. Association of Vitamin D deficiency with heart failure and sudden cardiac death in a large cross-sectional study of patients referred for coronary angiography. J Clin Endocrinol Metab. 2008 Oct;93(10):3927-35. Epub 2008 Aug 5.
26.) Wu PW, Rhew EY, Dyer AR, Dunlop DD, Langman CB, Price H, Sutton-Tyrrell K, McPherson DD, Edmundowicz D, Kondos GT, Ramsey-Goldman R. 25-hydroxyVitamin D and cardiovascular risk factors in women with systemic lupus erythematosus. Arthritis Rheum. 2009 Oct 15;61(10):1387-95.
27.) Baz-Hecht M, Goldfine AB. The impact of Vitamin D deficiency on diabetes and cardiovascular risk. Curr Opin Endocrinol Diabetes Obes. 2010 Apr;17(2):113-9.
28.) Cheng S, Massaro JM, Fox CS, Larson MG, Keyes MJ, McCabe EL, Robins SJ, O’Donnell CJ, Hoffmann U, Jacques PF, Booth SL, Vasan RS, Wolf M, Wang TJ. Adiposity, cardiometabolic risk, and Vitamin D status: the Framingham Heart Study. Diabetes. 2010 Jan;59(1):242-8. Epub 2009 Oct 15.
29.) Holick MF. Sunlight and Vitamin D for bone health and prevention of autoimmune diseases, cancers, and cardiovascular disease. Am J Clin Nutr. 2004 Dec;80(6 Suppl):1678S-88S.
30.) Ginde AA, Scragg R, Schwartz RS, Camargo CA Jr. Prospective study of serum 25-hydroxyVitamin D level, cardiovascular disease mortality, and all-cause mortality in older U.S. adults. J Am Geriatr Soc. 2009 Sep;57(9):1595-603. Epub 2009 Jun 22.
31.) Grant WB. How strong is the evidence that solar ultraviolet B and Vitamin D reduce the risk of cancer?: An examination using Hill’s criteria for causality. Dermatoendocrinol. 2009 Jan;1(1):17-24.
32.) Holick MF, Chen TC. Vitamin D deficiency: a worldwide problem with health consequences. Am J Clin Nutr. 2008 Apr;87(4):1080S-6S.
33.) Holick MF. Vitamin D: its role in cancer prevention and treatment. Prog Biophys Mol Biol. 2006 Sep;92(1):49-59. Epub 2006 Mar 10.
34.) Ingraham BA, Bragdon B, Nohe A. Molecular basis of the potential of Vitamin D to prevent cancer. Curr Med Res Opin. 2008 Jan;24(1):139-49.
35.) Pilz S, Dobnig H, Winklhofer-Roob B, Riedmüller G, Fischer JE, Seelhorst U, Wellnitz B, Boehm BO, März W. Low serum levels of 25-hydroxyVitamin D predict fatal cancer in patients referred to coronary angiography. Cancer Epidemiol Biomarkers Prev. 2008 May;17(5):1228-33. Epub 2008 May 7.
36.) Pilz S, Tomaschitz A, Obermayer-Pietsch B, Dobnig H, Pieber TR. Epidemiology of Vitamin D insufficiency and cancer mortality. Anticancer Res. 2009 Sep;29(9):3699-704.
37.) Ginde AA, Mansbach JM, Camargo CA Jr. Association between serum 25-hydroxyVitamin D level and upper respiratory tract infection in the Third National Health and Nutrition Examination Survey. Arch Intern Med. 2009 Feb 23;169(4):384-90.
38.) Annweiler C, Schott AM, Allali G, Bridenbaugh SA, Kressig RW, Allain P, Herrmann FR, Beauchet O. Association of Vitamin D deficiency with cognitive impairment in older women: cross-sectional study. Neurology. 2010 Jan 5;74(1):27-32. Epub 2009 Sep 30.
39.) Cherniack EP, Troen BR, Florez HJ, Roos BA, Levis S. Some new food for thought: the role of Vitamin D in the mental health of older adults. Curr Psychiatry Rep. 2009 Feb;11(1):12-9.
40.) Wilkins CH, Sheline YI, Roe CM, Birge SJ, Morris JC. Vitamin D deficiency is associated with low mood and worse cognitive performance in older adults. Am J Geriatr Psychiatry. 2006 Dec;14(12):1032-40.
41.) Cutolo M, Otsa K. Review: Vitamin D, immunity and lupus. Lupus. 2008;17(1):6-10.
42.) Kamen DL, Cooper GS, Bouali H, Shaftman SR, Hollis BW, Gilkeson GS. Vitamin D deficiency in systemic lupus erythematosus. Autoimmun Rev. 2006 Feb;5(2):114-7. Epub 2005 Jun 21.
43.) Bischoff-Ferrari HA, Willett WC, Wong JB, Giovannucci E, Dietrich T, Dawson-Hughes B. Fracture prevention with Vitamin D supplementation: a meta-analysis of randomized controlled trials. JAMA. 2005 May 11;293(18):2257-64.
44.) Bischoff HA, Stähelin HB, Tyndall A, Theiler R. Relationship between muscle strength and Vitamin D metabolites: are there therapeutic possibilities in the elderly? Z Rheumatol. 2000;59 Suppl 1:39-41.
45.) DIPART (Vitamin D Individual Patient Analysis of Randomized Trials) Group. Patient level pooled analysis of 68 500 patients from seven major Vitamin D fracture trials in US and Europe. BMJ. 2010 Jan 12;340:b5463. doi: 10.1136/bmj.b5463.
46.) Houston DK, Cesari M, Ferrucci L, Cherubini A, Maggio D, Bartali B, Johnson MA, Schwartz GG, Kritchevsky SB. Association between Vitamin D status and physical performance: the InCHIANTI study. J Gerontol A Biol Sci Med Sci. 2007 Apr;62(4):440-6.
47.) Kwon J, Suzuki T, Yoshida H, Kim H, Yoshida Y, Iwasa H. Concomitant lower serum albumin and Vitamin D levels are associated with decreased objective physical performance among Japanese community-dwelling elderly. Gerontology. 2007;53(5):322-8. Epub 2007 May 29.
48.) Pfeifer M, Begerow B, Minne HW. Vitamin D and muscle function. Osteoporos Int. 2002 Mar;13(3):187-94.
49.) Judd SE, Nanes MS, Ziegler TR, Wilson PW, Tangpricha V. Optimal Vitamin D status attenuates the age-associated increase in systolic blood pressure in white Americans: results from the third National Health and Nutrition Examination Survey. Am J Clin Nutr. 2008 Jan;87(1):136-41.
50.) Pilz S, Dobnig H, Fischer JE, Wellnitz B, Seelhorst U, Boehm BO, März W. Low Vitamin d levels predict stroke in patients referred to coronary angiography. Stroke. 2008 Sep;39(9):2611-3. Epub 2008 Jul 17.
51.) Melamed ML, Michos ED, Post W, Astor B.25-hydroxyVitamin D levels and the risk of mortality in the general population. Arch Intern Med. 2008 Aug 11;168(15):1629-37.
52.) Jacobs ET, Alberts DS, Foote JA, Green SB, Hollis BW, Yu Z, Martínez ME. Vitamin D insufficiency in southern Arizona. Am J Clin Nutr. 2008 Mar;87(3):608-13.
53.) Park S, Johnson MA. Living in low-latitude regions in the United States does not prevent poor Vitamin D status. Nutr Rev. 2005 Jun;63(6 Pt 1):203-9.
54.) Low Vitamin D Levels Tied to Incontinence. WebMD March 22, 2010 http://www.webmd.com/urinary-incontinence-oab/news/20100322/low-Vitamin-d-linked-incontinence.
55.) The Vitamin D Council. Vitamin D Council
56.) Wikipedia: Vitamin D. Wikipedia Vitamine D
57.) Holick MF. Environmental factors thatinfluence the cutaneous production of Vitamin D. Am J Clin Nutr. 1995 Mar;61(3 Suppl):638S-645S.
58.) Vieth R. Vitamin D supplementation, 25-hydroxyVitamin D concentrations, and safety. Am J Clin Nutr. 1999 May;69(5):842-56.
59.) Holick MF. Vitamin D deficiency. N Engl J Med. 2007;357:266-281.
60.) GrassrootsHealth. Disease incidence prevention by serum 25(OH)D level.Grassroots Heaalth. Accessed May 8, 2009.
61.) Dall T, Anderson J. Vitamin D: merging research into clinical lipid practice. Lipid Spin. 2008;6(3):4-8.
62.) Heaney RP. What is a Vitamin D deficiency?Grassroots Health Vitamin D deficiency. Accessed May 8, 2009.